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铜中毒/铝缺乏Copper Poisoning/Molybdenum DeficiencyPrepared by:Dr.Terry HuntTranslated byDr.傅小平病因/发病机理CausePathogenesis•牛少见,绵羊多见Not common in cattlebut morecommoninsheep•日粮中Cu/Mo超过10:1Cu:Mo rationis inexcess of10:1•绵羊用微量元素添加,平均值喂牛Supplement sheepwith traceminerals meantfor cattle危险因素Risk FactorsAnthelmintics containingcopper•含铜的抗蠕虫药Feed contaminationwith excessivecopper•饲料被铜污染Certain breedsof sheepare moresusceptible•某种绵羊易发病Clinical Findings临床症状•急性Acute•溶血肝中释放大量的铜Hemolytic crisismassive releaseof Cufrom liver♦贫血Anemia•血红蛋白尿Hemoglobinuria•黄疸Icterus•严重腹泻Severe diarrhea•慢性Chronic•长时间摄入大量的铜,无急性临床表现Intake ofCu overa longperiod oftime butacute symptoms断Diagnosis•临床症状、肝和血液中铜含量Clinical signsand Cu levels inblood andliver鉴别诊断Differential Diagnosis•细菌性血红蛋白尿Bacillary hemoglobinuriaPost-•产后血红蛋白尿parturient hemoglobinuria•急性钩端螺旋体Acute leptospirosis实验室和病理学诊断Laboratory andPathology•急性中毒铜含量不一定会高May notbe elevatedblood levelsif acutepoisoning•慢性中毒,铜含量超过2ppin Withchronic poisoning,Culevelsover2ppm•死后剖解一肝大面积坏死和黄疸Post mortem-massive livernecrosis andIcterus■红尿Port wineurine治疗Treatment•治疗比较困难,nJ试用螯合剂Treatment isdifficult butcan trychelating agent•添加硫酸钥和硫酸钠制剂Supplement withMolybdenum andSodium Sulfate预后情况Prognosis Poor•不良PreventionControl预防在含钥的草地放牧Top dresspastures withMolybdenum。